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This title is printed to order. This book may have been self-published. If so, we cannot guarantee the quality of the content. In the main most books will have gone through the editing process however some may not. We therefore suggest that you be aware of this before ordering this book. If in doubt check either the author or publisher’s details as we are unable to accept any returns unless they are faulty. Please contact us if you have any questions.
Periodontitis is an inflammatory disease harming teeth's support tissues due to specific microbes, causing the slow erosion of ligament and bone, resulting in pocket formation, recession, or both. Periodontal infection is initiated by specific invasive oral pathogens that colonize dental plaque biofilms on the tooth surface, and the host immune response to inflammation plays a central role in disease pathogenesis. There is an equilibrium that exists between microbial challenge and the host's immune response; any alteration to that with the addition of other modifying factors is responsible for the clinical manifestation of periodontal disease.
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This title is printed to order. This book may have been self-published. If so, we cannot guarantee the quality of the content. In the main most books will have gone through the editing process however some may not. We therefore suggest that you be aware of this before ordering this book. If in doubt check either the author or publisher’s details as we are unable to accept any returns unless they are faulty. Please contact us if you have any questions.
Periodontitis is an inflammatory disease harming teeth's support tissues due to specific microbes, causing the slow erosion of ligament and bone, resulting in pocket formation, recession, or both. Periodontal infection is initiated by specific invasive oral pathogens that colonize dental plaque biofilms on the tooth surface, and the host immune response to inflammation plays a central role in disease pathogenesis. There is an equilibrium that exists between microbial challenge and the host's immune response; any alteration to that with the addition of other modifying factors is responsible for the clinical manifestation of periodontal disease.